Exotic mesons from quantum chromodynamics with improved gluon and quark actions on the anisotropic lattice

Hybrid (exotic) mesons, which are important predictions of quantum chromodynamics (QCD), are states of quarks and anti-quarks bound by excited gluons. First principle lattice study of such states would help us understand the role of ``dynamical'' color in low energy QCD and provide valuable information for experimental search for these new particles. In this paper, we apply both improved gluon and quark actions to the hybrid mesons, which might be much more efficient than the previous works in reducing lattice spacing error and finite volume effect. Quenched simulations were done at $\beta=2.6$ and on a $\xi=3$ anisotropic $12^3\times36$ lattice using our PC cluster. We obtain $2013 \pm 26 \pm 71$ MeV for the mass of the $1^{-+}$ hybrid meson ${\bar q}qg$ in the light quark sector, and $4369 \pm 37 \pm 99$Mev in the charm quark sector; the mass splitting between the $1^{-+}$ hybrid meson ${\bar c}c g$ in the charm quark sector and the spin averaged S-wave charmonium mass is estimated to be $1302 \pm 37 \pm 99$ MeV. As a byproduct, we obtain $1438 \pm 32 \pm 57$ MeV for the mass of a P-wave $1^{++}$ ${\bar u}u$ or ${\bar d}d$ meson and $1499 \pm 28 \pm 65$ MeV for the mass of a P-wave $1^{++}$ ${\bar s}s$ meson, which are comparable to their experimental value 1426 MeV for the $f_1(1420)$ meson. The first error is statistical, and the second one is systematical. The mixing of the hybrid meson with a four quark state is also discussed.Comment: 12 pages, 3 figures. Published versio

SED-inferred properties and morphology of Lyman-break galaxies at z∼1z\sim 1 in the CDF-S

After carefully cross-identifying a previously discovered GALEX-selected Lyman Break Galaxy (LBG) candidates one-to-one with their optical counterparts in the field of the CDF-S, we re-estimate their photometric redshifts using multi-wavelength data from UV, optical to NIR. We refine a new updated sample of 383 LBGs at 0.7\la z \la 1.4. Most LBGs are classified as starburst and irregular types. Ages spread from several Myr to 1.5Gyr. Their dust-corrected star formation rates (SFRs) and stellar masses ($M_*$) are from 4\my to 220\my and from 2.3\times 10^8 \msun to 4 \times 10^{11} \msun. The rest-frame FUV luminosity function of LBGs are presented. LBGs of irregular types mainly distribute along the "main sequence" of star forming galaxies while most LBGs of starburst types locate in the starburst region. A "downsizing" effect is clearly found and LBGs distribute in the "blue" cloud. HST images in F606W ($V$ band) and F850LP ($z$ band) are taken from the GEMS and GOODS-S surveys. SExtractor and GALFIT are applied to get their morphological parameters. A morphological sample of 142 LBGs with reliable results of \sersic and sizes in both bands is defined. We find that LBGs at $z\sim 1$ are dominated by disk-like galaxies. Correlations between photometric and morphological properties of LBGs are investigated. Strong correlations between their half-light radii and $M_*$, i.e., size-stellar mass relations, are found in both bands. Physical connections between correlations and the "downsizing" effect are discussed.Comment: 26 pages, 30 figures, 6 tables, accepted by MNRA

Biochemical and genetic evidence for the involvement of yeast Ypt6-GTPase in protein retrieval to different Golgi compartments

Yeast Ypt6p, the homologue of the mammalian Rab6 GTPase, is not essential for cell viability. Based on previous studies with ypt6 deletion mutants, a regulatory role of the GTPase either in protein retrieval to the trans-Golgi network or in forward transport between the endoplasmic reticulum (ER) and early Golgi compartments was proposed. To assess better the primary role(s) of Ypt6p, temperature-sensitive ypt6 mutants were generated and analyzed biochemically and genetically. Defects in N-glycosylation of proteins passing the Golgi and of Golgi- resident glycosyltransferases as well as protein sorting defects in the trans-Golgi were recorded shortly after functional loss of Ypt6p. ER-to-Golgi transport and protein secretion were delayed but not interrupted. Mis-sorting of the vesicular SNARE Sec22p to the late Golgi was also observed. Combination of the ypt6-2 mutant allele with a number of mutants in forward and retrograde transport between ER, Golgi, and endosomes led to synthetic negative growth defects. The results obtained indicate that Ypt6p acts in endosome-to-Golgi, in intra-Golgi retrograde transport, and possibly also in Golgi-to-ER trafficking

B→DsπB \to D_s \pi and the tree amplitude in B→π+π−B \to \pi^+ \pi^-

The recently-observed decay $B^0 \to D_s^+ \pi^-$ is expected to proceed mainly by means of a tree amplitude in the factorization limit: $B^0 \to \pi^- {(W^+)}^*$, ${(W^+)}^* \to D_s^+$. Under this assumption, we predict the corresponding contribution of the tree amplitude to $B^0 \to \pi^+ \pi^-$. We indicate the needed improvements in data that will allow a useful estimate of this amplitude with errors comparable to those accompanying other methods. Since the factorization hypothesis for this process goes beyond that proved in most approaches, we also discuss independent tests of this hypothesis.Comment: 7 pages, LaTeX, 1 figure, to be submitted to Phys. Rev. D (Brief Reports

Adipose-derived stromal cells protect intervertebral disc cells in compression: implications for stem cell regenerative disc therapy

INTRODUCTION: Abnormal biomechanics plays a role in intervertebral disc degeneration. Adipose-derived stromal cells (ADSCs) have been implicated in disc integrity; however, their role in the setting of mechanical stimuli upon the disc's nucleus pulposus (NP) remains unknown. As such, the present study aimed to evaluate the influence of ADSCs upon NP cells in compressive load culture. METHODS: Human NP cells were cultured in compressive load at 3.0MPa for 48 hours with or without ADSCs co-culture (the ratio was 50:50). We used flow cytometry, live/dead staining and scanning electron microscopy (SEM) to evaluate cell death, and determined the expression of specific apoptotic pathways by characterizing the expression of activated caspases-3, -8 and -9. We further used real-time (RT-) PCR and immunostaining to determine the expression of the extracellular matrix (ECM), mediators of matrix degradation (e.g. MMPs, TIMPs and ADAMTSs), pro-inflammatory factors and NP cell phenotype markers. RESULTS: ADSCs inhibited human NP cell apoptosis via suppression of activated caspase-9 and caspase-3. Furthermore, ADSCs protected NP cells from the degradative effects of compressive load by significantly up-regulating the expression of ECM genes (SOX9, COL2A1 and ACAN), tissue inhibitors of metalloproteinases (TIMPs) genes (TIMP-1 and TIMP-2) and cytokeratin 8 (CK8) protein expression. Alternatively, ADSCs showed protective effect by inhibiting compressive load mediated increase of matrix metalloproteinases (MMPs; MMP-3 and MMP-13), disintegrin and metalloproteinase with thrombospondin motifs (ADAMTSs; ADAMTS-1 and 5), and pro-inflammatory factors (IL-1beta, IL-6, TGF-beta1 and TNF-alpha). CONCLUSIONS: Our study is the first in vitro study assessing the impact of ADSCs on NP cells in an un-physiological mechanical stimulation culture environment. Our study noted that ADSCs protect compressive load induced NP cell death and degradation by inhibition of activated caspase-9 and -3 activity; regulating ECM and modulator genes, suppressing pro-inflammatory factors and preserving CK8. Consequently, the protective impact of ADSCs found in this study provides an essential understanding and expands our knowledge as to the utility of ADSCs therapy for intervertebral disc regeneration.published_or_final_versio

Superconductivity at 41 K and its competition with spin-density-wave instability in layered CeO1−x_{1-x}Fx_xFeAs

A series of layered CeO$_{1-x}$F$_x$FeAs compounds with x=0 to 0.20 are synthesized by solid state reaction method. Similar to the LaOFeAs, the pure CeOFeAs shows a strong resistivity anomaly near 145 K, which was ascribed to the spin-density-wave instability. F-doping suppresses this instability and leads to the superconducting ground state. Most surprisingly, the superconducting transition temperature could reach as high as 41 K. The very high superconducting transition temperature strongly challenges the classic BCS theory based on the electron-phonon interaction. The very closeness of the superconducting phase to the spin-density-wave instability suggests that the magnetic fluctuations play a key role in the superconducting paring mechanism. The study also reveals that the Ce 4f electrons form local moments and ordered antiferromagnetically below 4 K, which could coexist with superconductivity.Comment: 4 pages, 5 figure

Fermi surface topology and low-lying quasiparticle structure of magnetically ordered Fe1+xTe

We report the first photoemission study of Fe1+xTe - the host compound of the newly discovered iron-chalcogenide superconductors. Our results reveal a pair of nearly electron- hole compensated Fermi pockets, strong Fermi velocity renormalization and an absence of a spin-density-wave gap. A shadow hole pocket is observed at the "X"-point of the Brillouin zone which is consistent with a long-range ordered magneto-structural groundstate. No signature of Fermi surface nesting instability associated with Q= pi(1/2, 1/2) is observed. Our results collectively reveal that the Fe1+xTe series is dramatically different from the undoped phases of the high Tc pnictides and likely harbor unusual mechanism for superconductivity and quantum magnetic order.Comment: 5 pages, 4 Figures; Submitted to Phys. Rev. Lett. (2009